COPD Pathogenesis

Inflammation is present in the lungs, especially the small airway routes, of all people who smoke. This typical defensive reaction to the inhaled toxins is enhanced in COPD, leading to tissue destruction, debilitation of the defence mechanisms and interruption of the repair mechanisms. In general, the inflammatory and structural changes in the airway routes increased with disease seriousness and hold on even after smoking cessation. Other than inflammation, two different procedures are associated with the pathogenesis of COPD—an irregularity amongst proteases and antiproteases and an imbalance amongst oxidants and antioxidants agents in the lungs.

  • Airway inflammation
  • Mucociliary dysfunction
  • Protease imbalance
  • Antiprotease imbalance

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